Search results for " glucose metabolism"

showing 10 items of 12 documents

p63 Isoforms Regulate Metabolism of Cancer Stem Cells

2014

p63 is an important regulator of epithelial development expressed in different variants containing (TA) or lacking (ΔN) the N-terminal transactivation domain. The different isoforms regulate stem-cell renewal and differentiation as well as cell senescence. Several studies indicate that p63 isoforms also play a role in cancer development; however, very little is known about the role played by p63 in regulating the cancer stem phenotype. Here we investigate the cellular signals regulated by TAp63 and ΔNp63 in a model of epithelial cancer stem cells. To this end, we used colon cancer stem cells, overexpressing either TAp63 or ΔNp63 isoforms, to carry out a proteomic study by chemical-labeling …

Gene isoformProteomicsProteomeRegulatorBiologyProteomicsBiochemistryTransactivationCancer stem cellmedicineHumansMetabolomicsProtein IsoformsProtein Interaction MapsSettore BIO/10 - BIOCHIMICAp63 colon cancer stem cells proteomics stable isotope dimethyl labeling glucose metabolismSettore BIO/12Tumor Suppressor ProteinsCancerGeneral Chemistrymedicine.diseasePhenotypePeptide FragmentsCell biologyIsotope LabelingNeoplastic Stem CellsStem cellSignal TransductionTranscription Factors
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Erratum: Effect of magnesium supplementation on glucose metabolism in people with or at risk of diabetes: a systematic review and meta-analysis of do…

2016

Although higher dietary intakes of magnesium (Mg) seem to correspond to lower diabetes incidence, research concerning Mg supplementation in people with or at risk of diabetes is limited. Thus, we aimed to investigate the effect of oral Mg supplementation on glucose and insulin-sensitivity parameters in participants with diabetes or at high risk of diabetes compared with placebo. A literature search in PubMed, EMBASE, SCOPUS, Cochrane Central Register of Controlled Trials and Clinicaltrials.gov without language restriction, was undertaken. Eligible studies were randomized controlled trials (RCTs) investigating the effect of oral Mg supplementation vs placebo in patients with diabetes or at h…

AdultBlood GlucoseMale0301 basic medicinemedicine.medical_specialtyCalorieMagnesium supplementationMedicine (miscellaneous)030209 endocrinology & metabolismCarbohydrate metabolismlaw.inventionDouble blind03 medical and health sciences0302 clinical medicineDouble-Blind MethodRandomized controlled triallawInternal medicineDiabetes mellitusDiabetes MellitusmedicineHumansMagnesiumAgedRandomized Controlled Trials as TopicGlucose tolerance test030109 nutrition & dieteticsNutrition and Dieteticsmedicine.diagnostic_testbusiness.industryMedicine (all)Medicine (miscellaneous); Medicine (all); Nutrition and DieteticsFastingGlucose Tolerance TestMiddle Agedmedicine.diseaseObesityEndocrinologyMeta-analysisDietary Supplementsmagnesium Nutrition diabetes Dietetics glucose metabolismFemalebusinessBody mass indexEuropean Journal of Clinical Nutrition
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Defective carbohydrate metabolism in multiple sclerosis

2015

La esclerosis múltiple (EM) es una enfermedad crónica del sistema nervioso central (SNC) en el que episodios repetidos de inflamación (bortes), dan lugar a inflamación que conduce a la interrupción de la vaina de mielina por daños producidos en la misma. Junto a este fenómeno de inflación focal, existe una inflamación difusa en el SNC, que unida a la anterior, dará lugar a que aparezca un proceso de neurodegeneración, que será el responsable último de la afectación axonal y neuronal difusa que es la que va a condicionar la discapacidad en los pacientes afectos de EM. La enfermedad es una causa importante de discapacidad neurológica y de disfunción neurológica en adultos jóvenes que afecta a…

Multiple Sclerosisneurodegenerative diseasebrain glucose metabolismIntermediate MetabolismMolecular Neurosciencerelapsing-remitting multiple sclerosiscerebrospinal fluid
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VEGF-targeted therapy stably modulates the glycolytic phenotype of tumor cells

2014

Abstract Anti-VEGF therapy perturbs tumor metabolism, severely impairing oxygen, glucose, and ATP levels. In this study, we investigated the effects of anti-VEGF therapy in multiple experimental tumor models that differ in their glycolytic phenotypes to gain insights into optimal modulation of the metabolic features of this therapy. Prolonged treatments induced vascular regression and necrosis in tumor xenograft models, with highly glycolytic tumors becoming treatment resistant more rapidly than poorly glycolytic tumors. By PET imaging, prolonged treatments yielded an increase in both hypoxic and proliferative regions of tumors. A selection for highly glycolytic cells was noted and this met…

Vascular Endothelial Growth Factor ACancer ResearchPathologymedicine.medical_specialtyNecrosismedicine.medical_treatmentAngiogenesis InhibitorsMice SCIDBiologySCIDAntibodies Monoclonal HumanizedAntibodiesCell LineTargeted therapyMiceRandom AllocationCell Line TumorNeoplasmsMonoclonalAngiogenesis Inhibitors; Animals; Antibodies Monoclonal Humanized; Bevacizumab; Cell Line Tumor; Female; Glycolysis; Humans; MCF-7 Cells; Mice; Mice Inbred BALB C; Mice SCID; Molecular Targeted Therapy; Neoplasms; Phenotype; Random Allocation; Vascular Endothelial Growth Factor A; Xenograft Model Antitumor AssaysmedicineAnimalsHumansGlycolysisMolecular Targeted Therapycancer-cellAnti-VEGF therapyHumanizedInbred BALB CMED/36 - DIAGNOSTICA PER IMMAGINI E RADIOTERAPIAMice Inbred BALB CTumorpositron emission tomography antiangiogenesis glucose metabolism hypoxiaXenograft Model Antitumor AssaysPhenotypeBlockadeBevacizumabVascular endothelial growth factor APhenotypeOncologyCell cultureMonoclonalMCF-7 CellsCancer researchMED/06 - ONCOLOGIA MEDICAFemalemedicine.symptomGlycolysis
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Perturbed Glucose Metabolism: Insights into Multiple Sclerosis Pathogenesis

2014

Multiple sclerosis (MS) is a complex debilitating disease of the central nervous system perceived to result from the autoimmune effect of T cells in damaging myelin sheath. However, the exact pathogenesis of the disease remains elusive. Initial studies describing the possibility of defective pyruvate metabolism in MS were performed in 1950s. The group observed elevated blood pyruvate level in both fasting and postprandial times in MS patients with relapse. Similarly, other investigators also reported increased fasting pyruvate level in this disease. These reports hint to a possible abnormality of pyruvate metabolism in MS patients. In addition, increase in levels of Krebs cycle acids like a…

medicine.medical_specialtyMini ReviewCentral nervous systemDiseaseCarbohydrate metabolismBioinformaticsmultiple sclerosislcsh:RC346-429PathogenesisInternal medicinebrain glucose metabolismmedicineneurodegenerative diseaseslcsh:Neurology. Diseases of the nervous systemmitochondrial defectsbusiness.industryMultiple sclerosisand neurodegenerative diseasesmedicine.diseasecell-specific mechanismsReview articleCitric acid cycleMetabolic pathwaymedicine.anatomical_structureEndocrinologyNeurologyNeurology (clinical)businessNeuroscienceFrontiers in Neurology
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Diabetes Secondary to Acromegaly: Physiopathology, Clinical Features and Effects of Treatment.

2018

Acromegaly is a rare disease due to chronic GH excess and to the consequent increase in IGF-1 levels. Both GH and IGF-1 play a role in intermediate metabolism affecting glucose homeostasis. Indeed, chronic GH excess impairs insulin sensitivity, increases gluconeogenesis, reduces the glucose uptake in adipose tissue and muscle and alters pancreatic β cells function. As a consequence, glucose metabolism alterations are a very frequent complication in acromegaly patients, further contributing to the increased cardiovascular risk and mortality. Treatment modalities of acromegaly differently impact on glucose tolerance. Successful surgical treatment of acromegaly ameliorates glucose metabolism a…

medicine.medical_specialtyGlucose uptakeMini Reviewglucose metabolismEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismCarbohydrate metabolismpituitary tumorlcsh:Diseases of the endocrine glands. Clinical endocrinologySettore MED/13 - EndocrinologiaImpaired glucose tolerance03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyInternal medicineDiabetes mellitusAcromegalymedicineGlucose homeostasislcsh:RC648-665diabetesbusiness.industrymedicine.diseasePasireotideGHEndocrinologyimpaired glucose toleranceAcromegaly Diabetes GH Glucose metabolism IGF-1 Impaired glucose tolerance Pituitary tumor Endocrinology Diabetes and Metabolismchemistry030220 oncology & carcinogenesisPegvisomantAcromegalyIGF-1businessmedicine.drug
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Physical Inactivity from Youth to Adulthood and Risk of Impaired Glucose Metabolism

2018

Introduction: Physical activity (PA) is important in the prevention and treatment of impaired glucose metabolism. However, association of physical inactivity during the transition between childhood and adulthood with glucose metabolism is unknown. Therefore, we studied the association of persistent physical inactivity since childhood with glucose metabolism in adulthood. Methods: Data were drawn from the ongoing, Cardiovascular Risk in Young Finns Study with repeated follow-ups between 1980 and 2011 (baseline age, 3-18 yr; n = 3596). Impaired glucose metabolism was defined as having impaired fasting glucose (6.1-6.9 mmol·L-1) or type 2 diabetes in adulthood. Leisure-time PA habits were repe…

Blood GlucoseMaleCross-sectional studyphysical activityType 2 diabetes030204 cardiovascular system & hematology0302 clinical medicineRisk FactorsSurveys and QuestionnairesOrthopedics and Sports MedicineLongitudinal Studies030212 general & internal medicineta315impaired glucose metabolismChildFinlandliikkumattomuusta3142Middle AgedaikuisuusChild PreschoolFemalefyysinen aktiivisuusAdultmedicine.medical_specialtyAdolescentPhysical Therapy Sports Therapy and RehabilitationpitkittäistutkimusCarbohydrate metabolism03 medical and health sciencesaineenvaihduntahäiriötInternal medicineDiabetes mellitusmedicineHumansExerciseLife Stylechildhoodbusiness.industryMetabolismlapsuusta3121medicine.diseaseImpaired fasting glucoseConfidence intervalCross-Sectional StudiesGlucoseEndocrinologyDiabetes Mellitus Type 2Relative riskphysical inactivitySedentary Behaviorbusinessaikuistyypin diabetesMedicine & Science in Sports & Exercise
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Exploring the Role of Skeletal Muscle in Insulin Resistance: Lessons from Cultured Cells to Animal Models

2021

Skeletal muscle is essential to maintain vital functions such as movement, breathing, and thermogenesis, and it is now recognized as an endocrine organ. Muscles release factors named myokines, which can regulate several physiological processes. Moreover, skeletal muscle is particularly important in maintaining body homeostasis, since it is responsible for more than 75% of all insulin-mediated glucose disposal. Alterations of skeletal muscle differentiation and function, with subsequent dysfunctional expression and secretion of myokines, play a key role in the pathogenesis of obesity, type 2 diabetes, and other metabolic diseases, finally leading to cardiometabolic complications. Hence, a de…

QH301-705.5glucose metabolismAdipose tissueReviewType 2 diabetesCarbohydrate metabolismMuscle DevelopmentBioinformaticsCatalysisInorganic ChemistryInsulin resistanceMyokineAnimalsHumansMedicinePhysical and Theoretical ChemistryBiology (General)Muscle SkeletalMolecular BiologyQD1-999Spectroscopybusiness.industryOrganic ChemistrySkeletal musclefree fatty acidsGeneral Medicinemedicine.diseaseComputer Science Applicationsadipose tissueglycemiaDisease Models AnimalChemistrymedicine.anatomical_structureDiabetes Mellitus Type 2Adipose tissue Free fatty acids Glucose metabolism Glycemia Myofibers Animals Diabetes Mellitus Type 2 Humans Muscle Skeletal Disease Models Animal Insulin Resistance Muscle DevelopmentInsulin ResistancebusinessThermogenesismyofibersHomeostasisInternational Journal of Molecular Sciences
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Disturbed Glucose Metabolism in Rat Neurons Exposed to Cerebrospinal Fluid Obtained from Multiple Sclerosis Subjects

2017

Axonal damage is widely accepted as a major cause of permanent functional disability in Multiple Sclerosis (MS). In relapsing-remitting MS, there is a possibility of remyelination by myelin producing cells and restoration of neurological function. The purpose of this study was to delineate the pathophysiological mechanisms underpinning axonal injury through hitherto unknown factors present in cerebrospinal fluid (CSF) that may regulate axonal damage, remyelinate the axon and make functional recovery possible. We employed primary cultures of rat unmyelinated cerebellar granule neurons and treated them with CSF obtained from MS and Neuromyelitis optica (NMO) patients. We performed microarray …

0301 basic medicinePathologymedicine.medical_specialtyglucose metabolismneuromyelitis opticaBiologymultiple sclerosisArticlecerebrospinal fluidlcsh:RC321-57103 medical and health sciencesMyelin0302 clinical medicineCerebrospinal fluidDownregulation and upregulationGene expressionmedicineRemyelinationAxonlcsh:Neurosciences. Biological psychiatry. Neuropsychiatrymultiple sclerosis; glucose metabolism; neuromyelitis optica; cerebrospinal fluid; gene expressionNeuromyelitis opticaGeneral NeuroscienceMultiple sclerosismedicine.disease030104 developmental biologymedicine.anatomical_structurenervous systemgene expression030217 neurology & neurosurgery
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Reduction in insulin sensitivity and inadequate β-cell capacity to counteract the increase in insulin resistance in children with idiopathic growth h…

2014

Purpose: To evaluate the performance of various indexes of insulin sensitivity and secretion and to identify the most useful indicator of deterioration of glucose metabolism in a cohort of children with growth hormone (GH) deficiency (GHD) during GH treatment. Methods: In 73 GHD children (55 M, 18 F; mean age 10.5 years) at baseline and after 12 months of treatment, we evaluated a number of surrogate indexes of insulin secretion and sensitivity. In a subgroup of 11 children we also performed an euglycemic hyperinsulinemic clamp. Results: After 12 months, a significant increase in fasting glucose (p < 0.001) and HbA1c levels (p < 0.001) was documented, despite all children remained with a no…

Blood GlucoseMalemedicine.medical_specialtyAdolescentHormone Replacement TherapyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentChildren; Glucose metabolism; Growth hormone treatment; Insulin secretion; Insulin sensitivity; Endocrinology Diabetes and Metabolism; EndocrinologyHypopituitarismCarbohydrate metabolismHypopituitarismSettore MED/13 - EndocrinologiaEndocrinologyInsulin resistanceInsulin-Secreting CellsInternal medicinemedicineHormone replacement therapy (male-to-female)HumansInsulinGrowth hormone treatmentChildDwarfism PituitaryChildrenGlucose metabolismGlucose tolerance testmedicine.diagnostic_testHuman Growth Hormonebusiness.industryInsulin secretionInsulinGlucose Tolerance TestInsulin sensitivitymedicine.diseaseGrowth hormone treatmentTreatment OutcomeEndocrinologyChild PreschoolConcomitantFemaleInsulin Resistancebusiness
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